Writing about love is like telling a story. It’s about sharing what’s in your heart and putting it into words. Love can be tricky; it makes us brave enough to speak up, but it can also hurt when the person we care about doesn’t feel the same way as we do. In this post, we’ll talk about how love is a mix of happy and sad moments, and how sometimes, e ven when we have to let go, love doesn’t really leave us. "Letting go" of someone we love is hard; it means being brave to accept the pain of losing someone you still care about deeply. Rejection hurts, leaving scars and pain that don't easily fade away. Like they say "Love completes a person", however, this same love also breaks a person too". Thinking about this deeply, this explains the dual nature of this profound emotion. We find ourselves immersed in a paradox where the very thing that brings us solace and joy can also be the same exact thing that inflicts the deepest wounds in our hearts, and in the c
Sickle cell anemia is a hereditary blood disorder that profoundly affects the lives of millions of individuals worldwide. This essay delves into the precise genetic and molecular underpinnings of sickle cell anemia, offering insights into how it occurs at the DNA level. Sickle cell anemia, also known as sickle cell disease, is caused by a point mutation in the gene responsible for hemoglobin production. Hemoglobin is a protein found in red blood cells that is crucial for transporting oxygen throughout the body. In healthy individuals, the hemoglobin gene (HBB) encodes for a protein called hemoglobin A (HbA), which consists of two alpha-globin chains and two beta-globin chains. The genetic mutation responsible for sickle cell anemia occurs at the DNA level within the beta-globin gene. The mutation results in the substitution of a single nucleotide base, adenine (A), for thymine (T) in the sixth codon of the gene, changing it from GAG to GTG. This single-point mutation leads to the repla